Diabetes
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Vergleich: Edelsteine.: Amethyst, Citrin, Mondstein, Smaragd.
Siehe: Anhang. (W. Karo/Farokh J.
Master/Anthroposofisch.) + Anhang 2 (Matthias
Girke) + Syndrom X + Krankheiten
+ Pancreas
Was
ist Diabetes?
Umgangssprachlich
Zuckerkrankheit genannt, bezeichnet der (nicht die) Diabetes mellitus vielfältige
Störungen des Stoffwechsels. Alle Betroffenen leiden unter chronischer
Hyperglykämie (Überzuckerung).
Die
Ursachen: Bestimmte Zellen der Bauchspeicheldrüse produzieren entweder kein
oder zu wenig des Hormons Insulin. Oder die Wirkung des Insulins auf wichtige
Organe wie Leber, Muskulatur und Fettgewebe ist gestört.
Insulin:
ein lebensnotwendiges Stoffwechselhormon, das den
Kohlehydrat-/Eiweiß-/Fettstoffwechsel steuert. Kann es nicht wirken, gerät der
Stoffwechsel aus der Balance.
(Quelle:
Deutsche Diabetes-Stiftung)
Diabetes
vom Typ 1 ist eine Autoimmunerkrankung. Das Immunsystem, das in erster Linie
Keime abwehrt, richtet sich dann gegen die Insulin produzierenden Zellen der
Bauchspeicheldrüse und zerstört diese. Die Insulinproduktion kommt zum
Erliegen. Die in der Nahrung enthaltenen Bau- und Brennstoffe (z.B.
Traubenzucker, also Glukose) können von den Körperzellen nicht mehr aufgenommen
werden.
Der
Typ-1-Diabetes zeigt sich oft schon im Kindes- und Jugendalter. Er macht aber
nur 5% aller Diabetiker aus!
Diabetes
vom Typ 2 ist eine Zivilisationskrankheit. Dabei ist die Wirkung des Insulins
in den Körperzellen vermindert, es besteht gleichzeitig ein Insulinmangel.
Diese
Erkrankung zeigt sich in unterschiedlichen Graden von Insulinresistenz und
-mangel. Der Typ-2-Diabetes oder die Vorstufen gehen oft mit anderen Anzeichen
des Metabolischen Syndroms einher - 80% der Betroffenen sind gleichzeitig stark
übergewichtig.
(Quelle:
Deutsche Diabetes-Stiftung)
[leider
ohne richtige Quelle, aber ein Versuch wert]
Nr.1
Medikament gegen Diabetes
Cannabis Oil
Ein
innovatives Arzneimittel für Diabeteskranke, entwickelt nach einer
amerikanischen Technologie
Normalisiert
den Blutzuckerspiegel
Reduziert
den Zuckergehalt
Hilft Insulin zu absorbieren
Verhindert
Hypoglykämie
Stärkt
das Immunsystem
Normalisiert
den Stoffwechsel
[Clifford J. Bailey]
Metaformin (dimethylbiguanide/Quelle remedia.at) has become the
preferred first-line oral blood glucose-lowering agent to manage type 2 diabetes.
Its history is linked
to Galega officinalis (= goat’s
rue), a traditional herbal medicine in Europe, found to be rich in guanidine in
1918, was shown to lower blood glucose.
Guanidine derivatives, incl.
metformin, were synthesised and some (not metformin) were used to treat
diabetes in the 1920s and 1930s but were discontinued due to
toxicity and the increased
availability of insulin. Metformin was rediscovered in the search for antimalarial/Coronavirusinfektion
= ähnlich
https://www.fixingdad.com/copy-of-home-1
[Dr. Sunirmal Sarkar
Kind: Crataegus is the only remedy in
our materia medica which has diabetes in children. It is also a well known
remedy for heart conditions.
[Masi-group and Jan Scholten]
Rosemary used as
additional therapy in diabetes
[Tinus Smits]
Carcinosin, Saccharum and Thuja
remedies for a family history of diabetes.
HYPOGLYCEMIA
In my practice I found several indications
for hypoglycemia or problems related to a disturbed sugar metabolism as:
ravenous appetite soon after eating; constant appetite, never satisfied;
sleepless > eating; tendency to eat frequently between meals; irresistible
desire to take sweets; strong impulse to eat on waking often because of a
fainting feeling or weakness; trembling when hungry; irritability when hungry
especially on waking, etc.
The great material cause of
hypoglycemia is without any doubt the exaggerated use of 'rapid' carbohydrates,
namely of refined sugar, in our modern nourishment.
The other material causes are coffee, alcohol and cigarettes.
The caffeine
in coffee, cola,
tea,
chocolate
and cocoa stimulates the adrenal glands to liberate adrenaline that stimulates
in its turn the release of sugar from the liver into the blood. About 70% of
alcohol abusers suffer from hypoglycemia. The sugar in the blood of smokers is
also increased after a cigarette. But there are also strong emotional causes
underneath such as lack of affection and inability to handle affection.
Hypoglycemia strongly influences our
social behavior and emotional well being. Dr. Michael Lesser stated that 67% of
his psychiatric patients suffered from hypoglycemia
and several researches show that our
mental and social behavior becomes erratic, violent and antisocial under
influence of hypoglycemia. And that is what I saw in many
of my patients with an unstable
sugar household (children): violent fits of anger with striking, kicking,
vandalism, aggressiveness and discontentedness.
Researchers discovered that the
behavior of juvenile delinquents is ameliorated if they are put on a sugar free
diet. In a study they found that 82% of a group of 106 suffered from
hypoglycemia. When changing their nutrition the majority ameliorated
dramatically their social behavior.(Spiritual nutrition and the rainbow diet;
Dr. Gabriel Cousens)
THE DIFFERENT MECHANISMS of
COMPENSATION for LACK of ATTENTION, AFFECTION and LOVE
In children we see more clearly the
compensation mechanisms. The most important are: a great need for cuddling, the
exaggerated sucking of fingers and the biting of nails,
(in adult life transformed in a
uncontrollable need to smoke), putting everything in the mouth and touching
everything. I often could verify the relation between the sucking
of fingers and the inveterate habit
of smoking, many patients admitting that they changed the first for the latter.
I was amazed to find so many adults who were still sucking their fingers. Many
people smoke to reduce or to control their weight, because they have
experienced already that stopping smoking means eating more, especially sweets.
Another mechanism frequently met is
loquacity, most patients being not aware of their secret demand for attention.
Children still have a lot of
possibilities for asking attention: doing pranks, asking again and again for
something or doing things that are forbidden, asking constantly for attention
when the parents talk to someone else and being jealous of their brother or
sister. All means can be used by the child that needs attention: being
restless, shouting, fighting, crying, having pain, being ill, etc.
In adults these mechanisms can
persist or change to more adult-like forms as the exaggerated need to possess
objects or to have new things with an everlasting feeling of dissatisfaction.
Also the incapacity to have a deep and lasting relation with someone, seeking
again and again for a new love affair, but never finding what he is really
seeking for. He is like a perforated bucket, you can put all the water you want
in it, but the bucket will empty itself constantly. There is a fundamental and
profound frustration from early life that cannot be satisfied at the level of
actual life. Only a deep transformation and cure can help such a person and
homeopathy can be a very effective tool for it.
[Veshara Malapermal]
Characterisation of diabetes
mellitus
Diabetes is one of the most common
metabolic disorders worldwide (Wild et al. 2004), characterised by a loss of
glucose homeostasis with disturbances in carbohydrate,
fat and protein metabolism resulting
from defects in insulin secretion, insulin action, or both (Katzung et al.
2009).
Without adequate insulin, body
tissues, in particular the liver, muscular and adipose tissues fail to take up
and use glucose from the blood circulation. The resultant elevated blood
glucose level is known as hyperglycaemia (Jarald et al. 2008).
Factors contributing to
hyperglycaemia:
Digestive System: Decreased incretin
effect
Kidneys: Increased glucosen
reabsorption
Liver: Increased hepatic
glucosenproduction and decreased hepatic glucose uptake
Stomach: Carbohydrates intake
Pancreas: Decreased insulin
secretion
Muscles: Decreased peripheral
glucose uptake
Islet alpha cell: Increased glucagon
secretion
Brain: Neurotransmitter dysfunction
Adipose tissue: Increased lipolysis
Diabetes occurs predominantly in two
forms, namely: Type 1 and Type 2.
These two forms differ in terms of
pathogenesis but produce essentially similar metabolic derangements (Nowak and
Handford 2004).
On the basis of aetiology, the term
Type 1 is widely used to describe insulin dependent diabetes mellitus (IDDM)
(Nowak and Handford 2004).
This type is the more severe of the
two forms, which typically is picked up at a young age, and is less common.
It develops following viral
infection, exposure to environmental chemicals, abuse of +/o. exposure to
therapeutic drugs or a strong genetic predisposition leading to antigen
alteration and subsequent immune attack, causing Beta-cell (β-cell)
destruction and leading to zero functioning of the β-cells, therefore
resulting in no secretion of insulin (Katzung et al.2009).
Type 2 diabetes mellitus, formerly
known as non-insulin dependent diabetes mellitus (NIDDM), is characterised by
chronic hyperglycaemia as a consequence of insulin deficiency caused by
insufficient synthesis or secretion of insulin from the β-cells, however
many contributing factors remain uncertain . (Nowak and Handford 2004).
Factors causing hyperglycaemia
Source: Katzung et al. 2009
Diabetes occurs predominantly in two forms, namely: Type 1 and Type 2.
These two forms differ in terms of pathogenesis but produce essentially
similar metabolic derangements (Nowak and Handford 2004).
On the basis of aetiology, the term Type 1 is widely used to describe
insulin dependent diabetes mellitus (IDDM) (Nowak and Handford 2004).
This type is the more severe of the two forms, which typically is picked
up at a young age, and is less common.
It develops following viral infection, exposure to environmental
chemicals, abuse of +/o. exposure to therapeutic drugs or a strong genetic
predisposition leading to antigen alteration and subsequent immune attack,
causing Beta-cell (β-cell) destruction and leading to
zero functioning of the β-cells, therefore resulting in no
secretion of insulin (Katzung et al. 2009).
Type 2 diabetes mellitus, formerly known as non-insulin dependent
diabetes mellitus (NIDDM), is characterised by chronic hyperglycaemia as a
consequence of insulin deficiency caused by insufficient synthesis or secretion
of insulin from the β-cells, however many contributing
factors remain uncertain (Nowak and Handford 2004).
Pathogenesis of NIDDM or Type 2
Adapted from:Nowak and Handford (2004) Type 2 diabetes is by far the
most common form of diabetes (Hannan et al. 2006), accounting for more than 90%
of population cases (Nguyen et al.2011).
This form of diabetes has a slow progression or development of symptom;
often years will pass without the victim being aware of any change (Nowak and
Handford 2004).
Insulin therapy is required less often in this type of diabetes, with
the exception being patients that fail to achieve proper glycaemic control,
during severe bacterial infections, ketoacidosis, during pregnancy and in
patients with impaired renal or hepatic function (Mizuno et al. 2008).
1.2.2
Complications of diabetes mellitus
Patients with diabetes experience
significant morbidity and mortality from micro- +/o. macrovascular
complications (Tayyab et al. 2012).
Microvascular disease is defined as
damage to the small blood vessels while macrovascular disease is damage to the
larger arteries (Nowak and Handford 2004).
The relationship between glycaemic
control and diabetic microvascular disease has been established in both Type 1
and Type 2 diabetes.
Acute complications include diabetic
ketoacidosis common to Type 1 diabetic patients, and non-ketotic hyperosmolar
coma, common in Type 2 diabetic patients (Tayyab et al. 2012).
Regardless of common diabetic
treatment regimens, chronic hyperglycaemia has been implicated as the main
cause of the adverse effects experienced by patients such as polydipsia,
polyphagia and lingering complications over a significant period (McCueet al.
2005).
The resultant long-term complications
are caused by damage to organs including the eyes, kidneys, nervous system and
blood vessels, causing various pathologies such as a therosclerotic vascular
disease (Ganet al. 1999/Bastaki 2005), ketoacidosis, nephropathy, neuropathy,
ulceration, eye complications [retinopathy = most common cause of blindness].
‘diabetic foot’ and limbamputation (Bastaki 2005/Lewis et al.2010).
Oxidative stress induced by chronic
hyperglycaemia (Kilet al. 2004) has been shown to be a major underlying
mechanism for the formation of harmful byproducts that accumulate and
contribute to development of the long-term complications associated with
diabetes (Neriet al. 2005).
Oxidative stress reflects an
imbalance between the systemic manifestations of reactive oxygen species (ROS)
constantly formed in the human body and the quantities of antioxidant products
required to restore balance, causing vasoconstriction (Kuyvenhoven and Meinders
1999). Excess production of ROS leads to the impairment of equilibrium between
pro-oxidants and antioxidant systems (Sharma and Kar 2014).
The activation of a number of
metabolic pathways induced by chronic hyperglycaemia produce end products that
contribute to the development of long-term complications associated with
diabetes (Kuyvenhoven and Meinders 1999). For example, the activation of the
polyol pathway causes decreased nitric oxide and prostaglandin synthesis, which
results in endothelial dysfunction and hypertension.
Increased polyol pathway activity
can induce retinopathy and neuropathy. Similarly, increased protein kinase C
(PKC) pathway activity and the formation of non-enzymatic
glycation of proteins can lead to
the increased risk of developing nephropathy, neuropathy and retinopathy.
In addition, increased hexosamine
pathway activity can potentiate macromolecular damage. Proper glycaemic
control, blood pressure management and lipid modification
are important to consider as they
may independently slow the progression of diabetic related micro- and
macrovascular complications, and thus reduce the rates of diabetic retinopathy,
neuropathy, nephropathy, diabetic foot infections, atherosclerosis and other
associated cardiovascular events, including dyslipidaemia, hypertension,
hypercoagulability and obesity (Ratner 2001/Mizuno et al. 2008).
1.2.3
Incidence of diabetes mellitus
Diabetes is a major health problem
with its frequency increasing every day from most developed and developing
countries (Wild et al. 2004). In South Africa, the prevalence is between 4% and
6%. The global prevalence was estimated at 2.8% in 2000 (171 million people
affected) (Wild et al.2004), 382 million people in 2013 and is estimated to
reach 592 million people by the year 2035, with a prevalence of 8.3%
(Guariguata et al. 2014).
WHO predicts that diabetes will be
the 7th leading cause of death by 2030 (WHO 2011).
The reasons for this global rise
have been linked to changes in lifestyle associated with urbanisation,
modernisation (Hamdan and Afifi 2004), growth of aged population, increasing trends
towards obesity, unhealthy diet, and sedentary lifestyles (Hannan et al. 2006).
Hypertension, coronary heart
disease, stroke, genetics and various forms of cancer are amongst the reasons
for the global rise in diabetes (Omran 1983/Reddy et al. 1998).
Obesity is a major concern because
it indirectly affects or exacerbates the incidence of diabetes, worsening the
severity of side effects or accelerating the incidence of diabetic related
complications (Mollentze and Levitt 2005/National Department of Health 2006).
These data shows that reliable, cost saving therapy is necessary to lower the
global rise of diabetes (Chiha et al. 2012).
Medicinal plants contain enormous
potential to provide alternative medicines for treating diabetes, but it is
necessary that their effectiveness is researched and substantiated.
The use of medicinal plants are part
of traditional practice in many countries and cultures (Soumyanath 2006)
including South Africa (van de Venter et al. 2008),
because of their availability, effectiveness,
minimal side effects and low cost.
Investigation into antidiabetic
agents from traditional medicinal plants is a major driver of research (Palatty
et al. 2013).
1.3
The use of traditional medicine in
the treatment of diabetes
In Africa, many plants are
traditionally used for the management and control of various ailments (Baynes
2006) incl. diabetes (Kavishankar et al. 2011).
Globally, approximately 85,000
medicinal plant species (sp.) are reported as medicinally useful (Liu and Wang
2008); however, few have received scientific scrutiny despite
medical and scientific
recommendation from the WHO (WHO 2007).
In South Africa, victims of chronic
diseases are turning to herbal medicines as alternative sources of treatment as
recommended by the National Department of Health (2006).
This renewed interest in plant
medicines as alternative therapy to restore health or treat diseases is
believed to be motivated by factors such as their effectiveness, that they are
more specific and that they contain diverse secondary metabolites which provide
numerous health benefits. The orchestra of chemical compounds within the plants
work together synergistically allowing active compounds to be available to
produce maximal therapeutic efficiency that are less toxic than high doses of
individual components (van Huyssteen 2007).
Traditional medicine (TM) may
provide an effective solution to the threat of diabetes worldwide, thus helping
to reduce chronic disease complications and deaths (Fang 2011).
World ethnobotanical information on
medicinal plants has reported up to 800 plants used for the treatment of
diabetes (Udayakumar et al. 2009).
Numerous medicinal plants offer
sustainable management of the sugar levels among diabetic patients and validated
for their hypoglycaemic potential using experimental animal models (Yeh et al.
2003).
Plants such as Momordica charantia
and Eugenia jambolana have been shown to ameliorate diabetic complications such
as neuropathy, nephropathy, fructose-induced insulin resistance, and cataracts
in experimental animals (Premila and Conboy 2007).
Diabetes is thus a common disease
for investigation using natural products. However, the mechanism of action +/o.
components that specifically exert blood glucose lowering effects on tissues or
organs remain unknown (Prabhakar and Doble 2011/Palatty et al. 2013).
Diabetic research on the therapeutic
effectiveness of natural plant products of South Africa is limited (Afolayan
and Sunmonu 2010).
Diabetes mellitus describes a metabolic
disorder of multiple aetiology characterized by chronic hyperglycaemia with
disturbances of carbohydrate, fat and protein metabolism
resulting from defects in insulin
secretion, insulin action, or both. The effects of diabetes mellitus include
long-term damage, dysfunction and failure of various organs.
Diabetes mellitus may present with
characteristic symptoms such as thirst, polyuria, blurring of vision, and
weight loss.
In its most severe forms,
ketoacidosis or a non-ketotic hyperosmolar state may develop and lead to
stupor, coma and, in the absence of effective treatment, death.
Often symptoms are not severe, or
may be absent, and consequently hyperglycaemia sufficient to cause pathological
and functional changes may be present for a long time before the diagnosis is
made (World Health Organisation, 1999: 2).
The long-term effects of diabetes
mellitus include progressive development of the specific complications of
retinopathy with potential blindness, nephropathy that may lead to renal
failure, +/o. neuropathy with risk of foot ulcers, amputation, Charcot joints,
and features of autonomic dysfunction, including sexual dysfunction.
People with diabetes are at
increased risk of cardiovascular, peripheral vascular and cerebrovascular
disease.
Several pathogenetic processes are
involved in the development of diabetes.
These include processes which
destroy the beta cells of the pancreas with consequent insulin deficiency, and
others that result in resistance to insulin action.
The abnormalities of carbohydrate,
fat and protein metabolism are due to deficient action of insulin on target
tissues resulting from insensitivity or lack of insulin (WHO, 1999:2-3).
Vipera berus
[Farokh Master]
The following homoeopathic medicine
has been extremely useful in my practice.
a) Cod.: excessive skin irritation (Itching
due to diabetes mellitus).
b) Ph-ac.: when diabetes
mellitus starts after some nervous exhaustion, working too hard or
disappointment in relationship.
c) Gun. Staph. Mela-a. extremely useful for diabetic carbuncle and gangrene.
d) Diabetic nephropathy.: Urea pura.
Ampe-q.
The treatment for diabetes mellitus
in homoeopathy is extremely effective but people do not come to us in early
state and hence once the pathology is advanced we cannot cure but only palliate
the condition.
On an average in my Homoeopathic
Health Centre I receive approximately 20 - 30 new patients every month with
high creatine level and uncontrolled diabetes mellitus. There patients have
been advised dialysis but with my experience of last 20 years I am able to
prevent dialysis in majority of these people with the help of constitutional
treatment and proper diet control.
I have been successful to arrest the
further progress of diabetic nephropathy. In our clinic we give advice for
diabetic diet:
a) Fruit and leaves of drumsticks (saijan
ki phalli) and patola are very useful for the diabetic patient.
b) Fenugreek (methi) seeds, when
given in varying doses of 25 grams to 100 grams daily, diminishes reactive
hyperglycemia in diabetic patient.
Soak 10 - 12 fenugreek (methi) seeds
in ¼ cup of water overnight and have both - the seeds and the water, next day
in the morning with a glass of water. This is beneficial for
people with diabetes. You can also
powder the methi seeds and take one teaspoonful of this powder with low fat
milk daily for at least two months. This will keep the blood
sugar level under control.
c) The diabetics should take two raw
string beans (runner beans or French beans) daily.
d) Have bhindi (okra) soup daily,
this will keep the diabetic patient healthy.
Method to prepare bhindi (okra)
soup:
Take 6 - 7 raw bhindis and wash them
thoroughly. Slit them lengthwise (do not cut the bhindi into two halves, just
slit them). Boil them in 1 litre of water till the water is reduced to half.
Strain this liquid and drink it plain
or with salt and pepper if you so desire. You mat throw away the boiled bhindi,
do not squeeze the bhindis while straining.
Having bhindi water is also useful,
the method is wash and cut 3 - 5 bhindis lengthwise in two pieces or in two
halves and soak them overnight in some water.
The next morning, remove the bhindis
from the water and drink that water. Doing this regularly will make the sugar
level normal within a couple of months.
[Florian
Schumann]
Cortisol
wird angewendet um Frühgeburten zu verzögern, löst Diabetes in Neugeborene in
später Zeitpunkt aus.
Clinical Features of Diabetes:
• Thirst
• Dry mouth
• Polyuria
• Nocturia
• Weight loss
• Pruritus vulvae
• Balanitis
• Fatigue/tiredness
• Myopia (Watkins et al., 2003:50).
Of Hippocrates' classic triad of
diabetic symptoms -polyuria, polydipsia, polyphagia- polyphagia is the most
intriguing, for this symptom most likely reflects the intracellular
(glucose deficiency) as opposed to
extracellular (glucose excess) pathophysiology of diabetes.
Hyperglycaemia does not, by itself,
entirely define the risk for diabetes or its complications.
Weight gain and insulin resistance,
of which excessive calorie intake is the first clinical sign, are the keys to
understanding type 2 diabetes (Fournier, 2000:603).
ORTHODOX PHARMACOLOGICAL
INTERVENTION
According to Huddle (1999:152), oral
hypoglycaemic agents are indicated in those patients with type 2 diabetes
mellitus who have not attained adequate glycaemic control despite adhering to
an effective programme of diet and exercise for four to six months, or who in
those patients who are symptomatic from the outset.
There are three major groups of oral
hypoglycaemic agents:
1.
Biguanide (e.g. Metformin : homöopatisch Quelle remedia.at)
Metformin is the initial therapy of
choice and should be initiated at the time of diagnosis in all patients (both
overweight and of normal weight), unless specifically contraindicated (SEMDSA,
2010:509).
Metformin acts by blocking glucose
production in the liver (gluconeogenesis) and may also stimulate tissue uptake
of glucose. It reduces insulin resistance but does not stimulate insulin
secretion (Whittaker, 2010:20).
Adverse effects of Metformin include
abdominal pain, bloating, diarrhoea, anorexia and very rarely lactic acidosis
(Whittaker, 2010:22). In the UK Prospective Diabetes Study
(UKPDS) it was found that Metformin
was responsible for a 42% reduction in diabetes related death (Whittaker,
2010:22). Metformin reduces HbA1C by 1 – 2% (SEMDSA, 2010:509).
2. Sulphonylureas (e.g.
Glibenclamide) Sulphonylureas are an option for first-line therapy when the
HbA1C is above target and the patient is normal weight; or the patient is
intolerant of Metformin; or when rapid control of hyperglycaemic symptoms is
needed (SEMDSA, 2010:509). The major mode of action of Sulphonylureas relies on
the ability of the pancreas to secrete insulin and they are therefore known as
insulin “secretagogues” (Whittaker, 2010:22-24).
Adverse effects of Sulphonylureas
include hypoglycaemia and weight gain (+ 2 kg) (SEMDSA, 2010:509).
According to Whittaker (2010:20-24),
Sulphonylureas have proven reduction of microvascular endpoints but unlike
Metformin, they have not produced significant reductions in myocardial
infarction, diabetes-related death and overall mortality. Sulphonylureas
reduces HbA1C by 1 - 2% (SEMDSA, 2010:509).
3. Thiazolidenediones (e.g.
Rosiglitazone) Thiazolidenediones are generic agents and are preferred because
of cost-effectiveness (SEMDSA, 2010:509). Thiazolidenediones
act by enhancing insulin action and
promoting glucose utilisation in peripheral tissues and suppressing
gluconeogenesis in the liver. They reduce insulin resistance but have no effect
on insulin secretion (Whittaker, 2010:24).
Adverse effects of
Thiazolidenediones include unpredictable weight gain, development of peripheral
oedema, mild anaemia and worsening heart failure (Whittaker, 2010:25).
Potential beneficial effects of
Thiazolidenediones include an improved cholesterol profile (Whittaker,
2010:24). Thiazolidendiones reduces HbA1C by 0.5 - 1.4% (SEMDSA, 2010:510).
Gluconol
[Dr. V. Krishnamurthy]
Most people think that as we have
remedies for diabetes in other systems (like insulin in allopathy) there would
be corresponding remedies in homeopathy. This is not so.
There is one marked difference
between homeopathy and other medical systems (allopathy, ayurveda, siddha)
In allopathy no doctor would give
insulin for typhoid; also no one would give antibiotic for diabetes. This is
because insulin is for diabetes and no amount of insulin will be of
any use in typhoid.
But in homeopathy one remedy given
for eczema patient may be indicated for jaundice in another; the same remedy
may be needed in the third patient suffering from cancer.
In such a case the question that
would naturally arise in the reader’s mind is, “If so, then what is the basis
for prescribing or selecting the remedy in homoeopath?” Answer to this
question will throw light on the
uniqueness of homeopathy.
[Munta]
Uran-n.: Laning states that no remedy gives
such universally good results; it lessens the sugar and quantity of the urine;
he recommended the 3x trituration. It is when the disease is
due to assimilative derangements that Uranium nitricum is the remedy, and
symptoms such as defective digestion, languor, debility and much sugar in the
urine, enormous appetite
and thirst, yet the patient continues to emaciate.
Syzyg.: Is capable of diminishing the amount of
sugar in the urine (used in the tincture and lower triturations).
Vermeulen (2000:1520), A most useful remedy in diabetes mellitus. No
other remedy causes in so marked degree the dimunition and disappearance of
sugar in the urine.
Ph-ac.: Corresponds to diabetes of nervous
origin. It suits cases due to grief and anxiety, those who are indifferent and
apathetic, poor in mental and physical force.
It is unquestionably curative of diabetes mellitus in the early stages, where
there is great debility and bruised feeling in the muscles.
There will be loss of appetite, sometimes unquenchable thirst and
perhaps the patient will be troubled with boils.
Large quantities of pale colorless urine or where there is much
phosphatic deposit in the urine it is the remedy.
Vermeulen (2000:1218): should be considered whenever the system has been
exposed to the ravages of acute disease, excesses, grief, and loss of vital
fluids.
Plb-met.: Hering considered Plumbum one of
the most important remedies in diabetes mellitus.
Vermeulen (2000:1269 - 1270), the diabetic symptoms of Plumbum
metallicum are great hunger or complete loss of hunger, unquenchable thirst
especially for cold water and scanty urine.
Bry.: no remedy has dryness of the lips as a
symptom of hepatic disorder more marked than Bryonia, and this is often one of
the first symptom of diabetes. There is a persistent bitter taste,
languid, morose and dispirited, thirst may not be extreme nor the
appetite voracious, may lose strength through inability to eat.
Momordica charatia
According to Leung et al. (2009:1702), bitter
melon is a popular fruit used for the treatment of diabetes amongst the
indigenous populations of Asia, S. America and East Africa.
The rationale for using Momordica charantia in
mother tincture and homoeopathic dilution to treat type 2 diabetes mellitus
despite it being unproved homoeopathically is that
Momordica charantia is a common food in Indian
cuisine and has been used extensively in folk medicine as a remedy for diabetes
(Kumar et al., 2010:95). Murray (1995:357
- 358) mentions that Momordica charantia is
composed of several compounds with confirmed anti-diabetic properties. Charantin,
extracted by alcohol, is a hypoglycaemic agent
composed of mixed steroids that is more potent
than the oral hypoglycaemic drug tolbutamide.
Momordica charantia also contains an
insulin-like polypeptide, polypeptide P, which lowers blood sugar levels when
injected subcutaneously into type 1 diabetics.
Adverse effects of Momordica charantia include:
- Hypoglycaemic coma
- Favism (In individuals with glucose - 6
phosphate defiency)
- Induction of abortions
- Abdominal discomfort
- Diarrhoea (Leung et al., 2009:1706).
The largest study, published in a 1999 issue of
the Bangladesh Medical Research Council Bulletin, used an aqueous suspension of
bitter melon vegetable pulp in 100 patients with type 2 diabetes mellitus. The
authors evaluated the effect 1 hour after bitter melon was administered and
then 2 hours after a
75 gram oral glucose tolerance test.
The average blood glucose was 222mg/dl
(12.33mmol/L), which was lower than the previous day’s 2-hour value of 257mg/dl
(14.28mmol/L) (Shane-McWhorter, 2005:2).
Cinnamon
A study conducted by Khan et al. (2003:3215)
demonstrated that the intake of 1 gram, 3 grams, or 6 grams of cinnamon per day
reduces serum glucose (18 – 29% after 40 days),
triglyceride, LDL cholesterol, and total cholesterol
in people with type 2 diabetes and suggest that the inclusion of cinnamon in
the diet of people with type 2 diabetes will reduce
risk factors associated with diabetes and
cardiovascular disease.
Gymnema sylvestre
Two non-randomized controlled clinical trials
involving groups of patients with type 1 diabetes and type 2 diabetes showed
improved glycaemic control with chronic adjunctive use of Gymnema sylvestre
(GS4) extract compared with those who received conventional treatment alone
(Yeh et al., 2003:1286).
Panax ginseng
The effects of Panax (Asian or Korean) ginseng,
given in a dosage of 100 or 200 mg per day for eight weeks, were studied in 36
patients with newly diagnosed
type 2 diabetes mellitus.
The study showed improved fasting blood glucose
levels.
The 200 - mg dose also resulted in improved
HbA1C levels (Kiefer and Pantuso, 2003:1541).
It is concluded by Vuksan and Sievenpiper
(2005:149) that the best evidence for clinical efficacy in diabetes remains for
ginseng.
Als diabetische Angiopathie werden Gefäßschädigungen bezeichnet, die als Spätkomplikationen bei Diabetes mellitus auftreten.
Durch Veränderungen der Kapillaren (Mikroangiopathie) werden verschiedene Formen der diabetischen Mikroangiopathie verursacht: insbesondere die diabetische Nephropathie, diabetische Retinopathie und diabetische autonome sowie sensorische und motorische Neuropathie.
Die Makroangiopathie ist zwar nicht Diabetes-spezifisch, tritt aber schneller und stärker als bei Nicht-Diabetikern auf, weshalb die Bezeichnung diabetische Makroangiopathie üblich ist. Sie führt zu atherosklerotischen Veränderungen an mittleren und großen Arterien. Sie ist für das hohe Herzinfarkt-, Schlaganfall- und Gangrän-Risiko bei Diabetes verantwortlich.
Die wichtigsten Faktoren, die die Entwicklung von Mikro- und Makroangiopathie bei Diabetikern fördern, sind Hyperglykämie, Hyperlipoproteinämie, Insulinresistenz sowie verschiedene vasoaktive Hormone, Cytokine und Wachstumsfaktoren.
Moschus.: Impotency associated with
diabetes.
Fungi
[Dr. Bradford]
In Diabetes, 2 or 3 lessened grains of the
3rd trituration of Uran-n. Administered morning and night, will in a
short time reduce the quantity of urine passed to nearly a normal standard, and
after a continual use, the proportion of sugar is materially.
[Lantz 2004]
Oploponax. (= Devil’s club) commonly
recommended for the treatment of type II adult onset diabetes, a use of devil’s
club that is also extensive in indigenous communities. . Since devil’s club is
still widely and increasingly, used as a treatment for late onset type II
diabetes and is listed in a recent review of anti-diabetic plants, additional
research and more rigorous clinical trials are required to validate and
characterise or to disprove hypoglycaemic properties in devil’s club.’
[Dr. L.R. Hughes]
Uran-n.:
diabetes originating in dyspepsia or assimilative derangement;
Ph-ac.:
starting point of the disease was in the nervous system.
[E.B. Nash]
Lac-ac.: Great remedy
for diabetes mellitus. Especially in addition to the thirst, voracious hunger
and profuse urine loaded with sugar, there are rheumatic pains in the joints.
High potency is much better and doesn’t not need frequent repetition.
[Wolfgang Mettler]
Thyr.: Diabetes mellitus - Gallensteine/mit Adipositas/mit Allergien in der Eigenanamnese, besonders beim Auftreten eines Diabetes nach spontanem Verschwinden
oder nach Unterdrückung allergischer Beschwerden wie z.B. Asthma, das periodisch auftrat.
Diabetes durch geistige Überanstrengung, durch Kummer (Ph-ac. Pic-ac. Sil. Tub.).
Rasche Entwicklung eines Diabetes mellitus mit extremer Schwäche.
[T.F. Allen]
Uran-n.: Numerous cases of diabetes are reported cured with this drug. Excessive
thirst, emaciation, and, perhaps, also tympanitis. Enormous appetite but the
patient emaciates.
www.joslin.org = amerikanische Website für Diabetiker
[Dr. rer. nat. Jan-Christoph Kattenstroth]
L-Arginin-Zufuhr kann Amputation der unteren Extremitäten verhindern
Mangeldurchblutung der Extremitäten im Zusammenspiel mit diabetesbedingten Nervenschädigungen ist die häufigste Ursache für das diabetische Fußsyndrom, eine der häufigsten Amputationsursachen in Deutschland. Eine im Jahr 2004 veröffentlichte Studie von Wissenschaftlern aus den USA konnte zeigen, dass L-Arginin der Mangeldurchblutung entgegenwirkt und so Amputationen verhindert werden können. Die Autoren weisen zusätzlich darauf hin, dass eine Zufuhr von L-Arginin zu einer deutlich schnelleren Abheilung der typischen Geschwüre führte – während Nebenwirkungen ausblieben (Arana et al., 2004).
Derzeit wird weiter viel an L-Arginin und den zugrundeliegenden Mechanismen bei Diabetes mellitus geforscht. Das beweisen drei aktuelle Studien aus den Jahren 2013 und 2014, die ebenfalls übereinstimmend zu dem Schluss kommen, dass eine ausreichende L-Arginin- Versorgung essenziell ist, um genügend von dem wichtigen Botenstoff Stickstoffmonoxid bilden zu können (Hoang et al., 2013; Rajapakse et al., 2013; Claybaugh et al., 2014). Da viele Diabetiker einen L-Arginin-Mangel aufweisen, setzen sie sich unnötig einem weiteren Risikofaktor für die gefürchteten Begleit- und Folgeerkrankungen des Diabetes mellitus aus (Robenek H, Poeggeler B, 2014).
Am besten in Kombination mit Folsäure, Vitamin B6 und B12
L-Arginin lässt sich eingeschränkt über die normale Nahrung aufnehmen. Natürliche Quellen sind vor allem Hülsenfrüchte und Nüsse. Bei Vorerkrankungen, wie Gefäßerkrankungen und Diabetes mellitus, ist ein höherer Arginin-Bedarf zu decken. Hierfür haben sich diätetische Lebensmittel in Form von Tabletten oder Pulvern mit L-Arginin bewährt. Damit L-Arginin und NO ihre Wirkung voll entfalten können, sollte auf die Kombination mit Folsäure, Vitamin B6 und Vitamin B12 geachtet werden (z.B. in „Telcor Arginin plus“, rezeptfrei, Apotheke). B-Vitamine unterstützen als
Co-Faktoren der NO-Synthese nicht nur die Freisetzung von NO aus L-Arginin (Bendall et al., 2014), sie spielen auch eine maßgebliche Rolle bei der Regulation und Senkung des Homocysteinspiegels im Blut. Homocystein ist eine schwefelhaltige Aminosäure, die über verschiedene Mechanismen die Verfügbarkeit von NO und L-Arginin im Körper verringert. Damit Homocystein im Körper abgebaut werden kann, müssen B-Vitamine in ausreichender Menge vorhanden sein (Martí-Carvajal et al., 2013).
[Paul Herscu]
Vipera: amputations, either accidental or else planned, due to underlying severe
chronic disease, such as uncontrolled diabetes. When diabetic patients have a
toe or two amputated due to damaged blood vessels, the recuperative time and
the complications can be extensive. It is then Vipera has worked well to
address the pain, swelling and mottled skin.
Fungi grow more actively in sugar solutions. Vaginal mycosis developes
quite often during pregnancy with its tendency towards pre-diabetes.
Krankheit: Juckreiz/schlecht heilende Hautbeschwerden, Starke Hypoglykämie: Schwindel/unsichere Gang/Ohnmacht/Schwitzen/Blässe/Zittern/Herz-klopfen/nervös/
Unterleibschmerz/ANfällig vor
Erkältung + Halsschmerz + Grippe + Bronchitis + Lungenentzündung (Polio);
Patient riecht wie Zucker
Die Erkrankungen des peripheren Nervensystems bei Diabetes mellitus = verschieden. Am häufigsten ist die längenabhängige distal-symmetrische sensomotorische Polyneuropathie manifestiert sich zunächst an längsten
Nervenfasern (Ischiasnerv), Früh: sockenförmig verteilte Parästhesien/Störung in Empfindungen auf Reizen. Später: Schmerz/Muskelkrämpfe/teilweise Lähmung der kleinen Fußmuskeln und Fuß- und Zehenheber
(oft nicht beachtet) sind neben Sensibilitätsstörung und autonomer Denervierung wegbereitend für das diabetischen Fußsyndrom. Differenzialdiagnosen: immunbedingte entzündliche Polyneuropathien mit und ohne
Paraproteinämie (falsche und zu viele Proteine im Blut), eine Radikuloneuritis [Wurzelentzündung der Nerv(en)] bei Borreliose und toxisch bedingte Neuropathien zu bedenken. Seltener Karpaltunnelsyndrom/örtliche
Neuropathien (Amyotrophie/Ophthalmoplegie).
Gemeinsames Merkmal dieser diabetischen Neuropathieformen = akutes neuropathisches Schmerzsyndrom, rasch gefolgt von umschriebener Muskellähmung.
Ursache: Pertussis vaccine has been connected
with juvenile onset diabetes as the vaccine acts directly on the islets of
Langerhans, the insulin-secreting parts of the pancreas (Gaublomme, 1997).
1. hypoglycæmia 2. diabetes.
This emphasises the link between candidiasis and diabetes, as does the
fact that the organism is identified by the physiological character of its
assimilation and fermentation
of sugar. The use of Foll. successfully in candidiasis (Assilem, 1990)
similarly underlines the connection with female sex hormones;
Blutzucker nüchtern vor Frühstück 80 - 90 Mg
Blutzucker in 100 Ml im Blut/ (nicht
> 130)
Nach 2 h. nach Zuckerhaltiges 120 - 140 Mg/> als 180 Mg = Hyperglycämie (Diabetes).
Nach 3e - 4e h. nach Zuckerhaltiges 80 - 90 Mg.. < 80 Mg. = Hypoglykämie (Kopfschmerz/schwach/Muskelmüdigkeit/Hunger/reizbar/nervöse Unruhe
Erhöhte Blutzuckerspiegel fördert Krebs.
Erniedrigte Blutzuckerspiegel kann Viren hemmen o. fördern
Karotte senkt Zuckergehalt wenn verursacht durch Mangel an Kali-salzen.
Vitamin B Komplex
Helon.: 1st stage,
urine profuse, clear, saccharin, lips dry stick together, THIRST, restless,
emaciation.
Kali-acet. in diabetes
Ign.. diabetes following emotional shock.
Vanad-s. = Katalysator zu Insulin/zu niedrigem Blutzuckergehalt,
Bromelain
Cyclopia intermedia Diabetes/Menopause
Cecr.
Galega officinalis. Blutzucker senkend
Grifolia frondosa Bluthochdruck/Krebstherapie/Blutzucker
senkend/Osteoporose/Leber
Lentinus edodes Diabetes Bindegewebe
stärkend, = appetithemmend/Ausdauer erhöhend/Fördert Umwandlung LDL-Chol. in HDL-Chol.
Aktiviert Immunsystem, hemmt Tumoren, steigert Ins.
Quelle: MykoVital GmbH 06047/7073
Remedies: Syzyg. Uran-n.
Ph-ac. Nat-m. Ceph. Gymne. Abroma-a. Aven. With-s. (= Ashwagandha). Helon.
Diabetes
Calc-ars. C 6.
Pancreatinum 3x. Cean. 3x. These 3 taken 3x daily will give good result. Covers
all types of diabetic.
1/4 teaspoonful Cinnamon powder takes as a tea or in food also helps to
reduce BS level.
Arn. C 6 as Wet. Dose.
--------- + Furunkel/+ Karbunkel: Cephd-i. Crot-h.
--------- + Gallensteinen But-ac
--------- + Gangrän: Colos. Sec. + Ars. + Kreos.
--------- + durst: Chion
--------- + rheumatic pain: Lac-ac.
--------- + schlaflos: Carc.
--------- + Schleimabsonderung Brust: Squil.
--------- + Nierenentzündung: Canthin
--------- Benz-ac + Ph-ac + Sal-ac.
Sil. + Silikaten?
Sulph. In the
beginning of diabetes.
Diabetes mellitus: Abrom-a-rx. Ars-br. Syzyg. Uran-n.
Diabetes Kind
---------------- Crat.
[Dr. Herbert A. Roberts]
Probably the type of glandular imbalance we meet most frequently is
diabetes mellitus. The accepted therapy is insulin, and it has a definite
influence on the sugar output; yet few physicians pause to consider whether
this treatment is curative or merely palliative - a substitution therapy.
Recent experiments indicate that continued massive doses of insulin may result
in an increase of sugar following an initial decrease; and that it may remain
at a fairly high level so long as the insulin therapy is pushed.
A case recently observed provided the interesting phenomenon of a marked
decrease of sugar output when the patient was forced to do without her insulin
for a
few days; and that when she returned to a decreased insulin dosage the
amount of sugar remained at a much lower level than while she was receiving
massive doses.
A series of observations on patients under homoeopathic care would be valuable.
We must remember that once insulin therapy is established, it tends to
become necessary to the patient and there is little hope of establishing normal
balance. Therefore it is more practical to begin treatment by the use of the
homoeopathic remedy, for we can always go to insulin later if this is
necessary. We find suitable remedies for Sugar in Urine in the repertories, and
most of the remedies listed are deep in action or are closely related to
emotional states. The diabetic patient usually presents subjective symptoms
that clearly indicate the simillimum, or he may be able to give a history of
emotional shock preceding his present affliction that will point the way to the
remedy. It is possible that his symptoms are so clearly marked that the indications
for a constitutional remedy cannot be overlooked, even though his remedy has
not been proven to produce the sugar imbalance.
In such case, if the patient improves on the indicated remedy, we are
justified in adding it to those already listed, giving it a tentative clinical
rating. If the general level of health is raised, even though the low sugar
threshold remains the same, we may safely rely on the remedy which maintains
general improvement, and not be too anxious over the sugar output. Recent research
work has indicated the influence of the pancreas in peptic ulcer.
Nahrungsergänzungsmittel: Ca/Cu/Ch/K/Se/Va/Mn/Zn/Mg/Vit B1/B6/Biotin/C/E//B12/Folic acid/ Ser-ang während Dialyse gebraucht verkürzt de Zeit der Dialyse.
[Alan Tillotson] From a blog.
I am a juvenile diabetic, insulin dependant, and have been so since age
10. I'm now 46. I have managed to avoid all complications so far, with perfect
retinas, normal heart, liver and kidney function etc. I got involved with
healing to try to find a way out of the bleak future I read about when I was 11
in a book I got from the library, which told me that I would most probably
develop any number of serious problems. So far zilch. I'm keeping my fingers
crossed. It is very interesting that before I had any formal training in
natural medicine, my body figured out several things (because it made me feel
good), such as the addition of lots of blueberries and raspberries
and carrots to my diet was good, as well as onion and garlic.
Answer:
Juvenile diabetics can learn to sense their own sugar levels by
developing body awareness. This makes it much less likely to eat when sugar
levels exceed 200, thus driving them up to the 300's and creating lots of
ketones, or to allow low blood sugars, with the negative effect of eating too
much too fast and having an upswing.
Juvenile diabetics which are called "brittle" are just
nutrient deficient or very nervous/tight. I have maintained a high nutrient diet
(easily assimilable minerals), for decades, and blood sugar swings have been
minimal. This includes multi-minerals supplements +/o. herbs like dandelion,
turmeric, nettles, parsley, seaweeds.
Juvenile diabetics need to constantly move their bodies. Hard work is
excellent medicine.
Juvenile diabetics tend to be vata natured (Ayurvedic term for nervous
personality), and so their diet needs more good quality oils and fats than
adult onset diabetics, to maintain healthy membranes.
The Ayurvedic combination of triphala (three fruits, famous ayurvedic
formula) plus shilajatu (ayurvedic mineral substance), taken long term,
prevents deterioration.
Some sort of Yoga or T'ai chi or meditation practice is essential to
develop the ability to get really relaxed (I mean like super, super relaxed),
which really helps maintain solid health, stable blood sugars. Advanced
pranayama or qi kung exercises, in which qi is made to move or flow with strong
diaphramatic control, improves the general circulation a lot.
Aspartame sometimes raises blood sugar in diabetics worse than pure cane
sugar.
San qi (Chinese herb) can arrrest hemorrhage in diabetic retinopathy
better than anything. 1-2 grams of concentrated powder TID, even useful long
term.
Blood thinning therapy (moving blood in Chinese terms) is essential
(Salv./Paeon./carthamus, cnidium, bilberry, turmeric, bromelain. Keeps the tiny
vessels happy, especially when combined with the minerals.
I advocate not worrying about money for those damn expensive test
strips, and just investing in other commodities. Juvenile diabetics tend to
have periods where their blood sugars become unstable, often for days or weeks.
During these times it is essential to check the sugar every few hours,
and take extra regular insulin if necessary, drink more water, and change to a
lower volume high protein and fat, low carbohydrate and sugar-free diet for a
short while, to even out sugar delivery to the system. When sugars are stable,
change to a more high vegetable carbohydrate and raw vegetable diet to even
things out. This alternation needs to be clearly understood.
Juvenile diabetics need more vitamin E. They do not seem to benefit from
chromium as much as adult onset types.
Lucillia sericata. = Made Schmeißfliege/scheidet in äußerliche Schleimhaut Allant. aus in Wunde (heilt Wunde), verzehrt nur krankes Gewebe/werden gebraucht um nicht schließende Wunden zu heilen. Quelle: Apotheke
Tupelohonig (gewonnen aus Nyssa sylvatica Cornales./= antibiotisch/= geeignet für Diabetiker).
Suis-pan. = Pankreas vom Schweineembryo
Vergleich: Lacs allgemein.; Siehe: Saccharum officinarum + Enzymen + Insulinum
Allerlei: Hypoglycemia = zu niedrige Blutzuckerspiegel/= Vorstufe Diabetes
Beschwerden: Nervös/Herzrasen/zittern/Schweiß/Denken erschwert/leichtes Empfinden im Kopf/Wirkung von Exzitotoxine
Syndrom X: = Beschwerden durch Insulinreistenz (Vorstufe Diabetes Type II).
1. Insulinreistenz + 2. ZU wenig Prog + 3. VIEL Eikosanoiden-2 verursachen in Wechseljahren. GEFÄHRLICHE Wechselwirkung
Vorbeugung: 3 große Mahlzeiten + 3 kleine Zwischenmahlzeiten aus HOCHwertige Nahrungsmittel (wenig Eiweiß aus Eier/Milchprodukten/Soja)
Verzichten auf raffinierten Kohlenhydraten/Süßes/Alkohol (steigert Östrog.-wirkung).
Defekte in der Insulin Sekretion zeigen sich in einer Hyperglykämie, welche besser unter Diabetes mellitus bekannt ist.
Type I: Insulinmangel durch verminderte Insulinausschüttung des Pankreas/= insulinabhängige Diabetes/fast nur Kinder und Jugendliche/wird den Autoimmunerkrankungen zugerechnet. Hier führt die Zerstörung der pankreatischen Inselzellen zu einem absoluten Insulinmangel. 5 - 10 % aller Erkrankten/Symptome wie Polyurie, Polydipsie und starker Gewichtsverlust sind charakteristisch.
Type II: Körper vermindert Insulinrezeptoren an Zellen, nimmt dadurch verfügbare Stärke nicht auf/= Altersdiabetes/Patienten ab einem Alter von 40 Jahren auf o. bei übergewichtigen Patienten. 90 - 95% aller Erkrankten. Die auftretenden Symptome sind minimal. Ein relativer Insulinmangel entsteht durch jahrelange Glucose-Überernährung, wodurch es zu einer Erschöpfung. der B-Zellen der Pankreas kommt.
Diagnose
Bei einem funktionierenden Zuckerstoffwechsel liegen die Blutglucosespiegel immer relativ konstant bei 70 bis 150 mg/dl. Chronisch erhöhte Glucosekonzentrationen im Plasma führen zu mikrovaskulären Krankheiten, wie die Retinopathie, verursachen Nierenfunktionsstörungen oder schädigen Nerven.
Insulin und C-Peptid
Ein erhöhter Blutzuckerspiegel stimuliert die B-Zellen der Bauchspeicheldrüse zur Produktion und Freisetzung von Insulin. Insulin erzeugt einen hypoglykämischen Effekt, indem es die zelluläre Aufnahme von Glukose in Muskel- und Fettgewebe fördert und die Glukoneogenese stoppt. Extrazelluläre Glukose wird durch Insulin zu den intrazellulären Speicherplätzen befördert, wo es in Form von Glykogen gespeichert wird. Glukagon, Epinephrin, GH und Cortisol wirken als Gegenspieler von Insulin. Sie erhöhen die Blutzuckerkonzentration indem sie die Glykoneogenese stimulieren.
Die Bestimmung von Insulin hat aber einen großen Nachteil. Die Insulintests können zwischen endogenem und exogenem Insulin nicht unterscheiden. Viele Patienten entwickeln Antikörper gegen Insulin, welche die Tests stören. Diese Problematik kann durch die Bestimmung von C-Peptid ausgeschaltet werden.
Indications for hypoglycemia/disturbed sugar metabolism: ravenous
appetite soon after eating/constant appetite/never satisfied/sleepless/>
eating/tendency to eat frequently between meals/irresistible desire to take
sweets/strong impulse to eat on waking often because of a fainting feeling or
weakness/trembling when hungry/irritable when hungry (on waking), etc.
Cause of hypoglycemia is USE of 'rapid' carbohydrates (refined
sugar/refined flour) in modern nourishment. Other causes are: caffeine (in
coffee/cola/tea/chocolate/cocoa)/
alcohol/cigarettes. Kaffein stimulates the adrenal glands to set
adrenaline free that stimulates in turn the release of sugar from the liver
into the bloodstream. Emotional causes
underneath such as lack of affection and inability to handle affection.
Hypoglycemia influences our social behavior/emotional well being. mental and
social erratic/violent/antisocial
behavior (child: violent fits of anger with striking, kicking,
vandalism, aggressive/discontent).
Behavior of juvenile delinquents > wenn put on a sugar free diet.
(Spiritual nutrition and the rainbow diet, Dr. Gabriel Cousens)
Compensation of lack of love/affection: getting it by any means or
refusal of any form (Lac-h.).
Opposite symptoms:
insatiable appetite (boulimia) versus
no appetite at all and sometimes a refusal to eat resulting in anorexia.
Thirst for large quantities versus no thirst at all.
Great need to cuddle versus refusing every contact.
Exaggerated sucking of fingers versus
no sucking of fingers at all,
putting everything in the mouth versus
never putting anything in the mouth.
Sensitive to pain versus
almost insensitive to pain.
No appetite in the morning versus
ravenous appetite on waking so that he has to eat first.
gentleness # very aggressive
behavior.
Cold feet, but sometimes so hot at
night sticking them out of bed.
The most frequent way to compensate a lack of affection seems to be by
far the use of sweets of any kind. A link between love and sweets ("sweet
boy or girl" "honey", "sweetheart", "sugar",
etc).
Many symptoms related to food desires and aversions, appetite and eating.
DESIRES any kind of sweets (chocolate/licorice/pastries/biscuits/“junk
sweets“).
Increased appetite before menses/desires sweets after dinner.
No appetite at all in the morning after rising and increased appetite in
the afternoon (evening/candies) and no need during the day.
Hypoglycemia with NEED to eat first on waking and tendency to eat
frequently between the meals, not feeling well if the meal is postponed a
little.
Feel weak/trembling/empty in the stomach/dizzy or get a headache if they
cannot eat at regular hours. Feel weak and irritable in the morning/difficult
to activate themselves/generally > after breakfast.
Neue Therapien:
Helo-s.x Gilatide (= peptide in saliva has shown
effect in the treatment of Alzheimer/Diabetes/ADD)/improves memory and
learning: Novel Nootropic (= smart drugs/memory enhancers/cognitive enhancers)
Peptide (= Exenatide®).
Verwandt sind Alzheimer und ALS.
Folgen bei Kinder von Mütter mit Diabetes: Puls = schwach + schnelles, vergrößertes Herz
Phytologie: Phlor. senkt Blutzuckerspiegel/‡ Rosm ‡
Datisca
cannabina = Scheinhanf/Cucurbitales .C 6 in Diabetes
‡ Ich-organisation / Sulphur indicated for vacillating blood sugar levels
with the typical sudden sweet-
craving and associated aggravation
of behavior; R.S.: Tannenharz der Signatur entsprechend bei Zuckerkrankheit
(Diabetes), die zu den typischen Altersleiden gehört und stets mit schlechter
Wundheilung einhergeht. ‡
‡ Folgendes hat anthroposofische Einschlüße ‡
R.S. explained in the 1920s: “Imagine that you are stressing the memory capacity
of the child excessively around the 9th or 10th year of
life, that memory is used too much as in education.
The consequences of this will show themselves only when the human being
is in his thirties or forties or even later. Then the person will become either
a sufferer of rheuma or diabetes. Precisely when memory is used inappropriately
around the 9th or 10nth year of life, then this overwhelming of memory in
childhood will show itself later in excessive deposits of metabolic products
... On the other hand, when the child is required to use too little memory
(appealing too little to the child’s ability to remember) then we will call
forth in later life a tendency for inflammatory processes of all sorts. To
understand how the bodily conditions of one epoch of life are the consequences
of the spiritual-soul conditions of another life-stage is one of the most
important things that we must realize”.
We are practicing plenty of that in the rearing of our children.
Over-intellectualization, unreasonable expectations of mental performance, now
often beginning with kindergarten, leads to the metabolic syndrome on the one
hand. And on the other hand, lack of a rhythmic lifestyle, starting with
irregular meals/irregular sleep times/stresses and anxieties for children
living between multiple families, can lead to the later life inflammations. Not
surprisingly, underlying obesity, as a fat deposit, and diabetes, as a sugar
deposit, scientists are finding a pervasive inflammatory process.
Ultimately the excesses of childhood reveal themselves as leading to a
generalized state of exhaustion of the Ego (the personal individuality) where,
sadly enough, this results in a culture where a majority of adults begin their
mature years in a chronically overwhelmed state [literally not taking hold of
our bodies (let alone of the social requirements around us)] and these untended
physiological processes fall apart and result in disease. It is a scenario like
this that points out the true incalculable importance of preventive hygiene and
holistic lifestyle changes.
Treatment of diabetes referring to medications or nutritional changes,
needs to focus on strengthening the Ego in addition to attempting to affect the
physical body in a more narrow sense by lowering sugar or burning fat. Inspired
by R.S.’s work there are several creative modalities that we can use along with
conventional therapies. The common denominator for all these ideas is a
reawakening of the personal individuality.
A classical indication given by R.S. is the use of Rosm. Initially he
recommended baths with a strong amount of rosemary oil with the admonition that
it should be clearly smelled by the patient so that one would be aware of the
herb through one’s sense organs during the bath. Various modalities have been
developed over the years to make this application more effective (oil
dispersion bath). The rosemary plant exhibits the principles that we are
considering above in an exemplary fashion. It takes the forces of fragrance and
warmth contained in its etheric oils back into the plant instead
of dispersing them generally into the surrounding environment the way
lavender does, for example. The plant does not allow its life-forces to be used
in developing large leaves or flowers. But rather, it concentrates all its
strength towards the inside and results in the typical woody, small, shrubby
appearance that is so characteristic of this plant. Interestingly Rosm. has
been found to be a powerful anti-inflammatory and anti-oxidant. Research has
shown, however, that it is ineffective if it doesn’t have an appropriate
quality control shown in the standardization of its most active ingredients of
Rosmarinic and Carnosinic acids. These new insight make it reasonable to use Rosm.
as an oral nutritional supplement also.
For Ego-strengthening, salt scrubs can be used to invigorate the entire
periphery. Often the salt is mixed with rosemary as a fragrance and its effect
is thus considerably amplified.
To overcome the chronic exhaustion of life forces stemming form the
above-mentioned educational “headyness,” remedies made from roots are very
helpful. While it is beyond the scope of this brief article to fully explain
the connection, it can be intuitively seen that there is a correspondence
between the herbal root system that connects the plant with the surrounding
earth and the human nerve-sense organization that connects us in turn with our
surrounding world. Root extracts have been known since antiquity as valued
tonics or as specific healers for various conditions. A gentian or curcumin
tincture help through their spiciness and bitterness to overcome stagnant
intestinal processes, particularly such common conditions accompanying diabetes
as an overly slow emptying of the stomach. More recently, other root
formulations from plants such as Withania or Rhodiola have been shown to be
particularly energizing and restorative.
Anthroposophical medicine has emphasis on with various forms of quartz =
silica. Silicates help not only in restoring the normal function of sense
organs that can be damaged by abnormal glucose levels but also help in
normalizing the metabolism of the sugar overload.
Several substances are involved in helping the Ego either connect itself
more strongly with the body or helping it to relax and go to sleep. If Calcium
in general can be seen as having more calming effects. It can be used against
allergies and sleeplessness. Phos. has an invigorating, awakening effect. In
the proper formulation, in a very dilute form, Phos. can help the spiritual
self to better take hold of metabolic processes.
Visc-a.: in phytology used to lower blood pressure/enhances fertility/also occasionally
used in the treatment of epilepsy. R.S.: prepared properly and given in
injectable form it had cancer fighting abilities. The mistletoe demonstrates
through its peculiar growth rhythms that it emphasizes at all times its own
“individuality.” Contrary to most other plants, it has a dormant state in the
summer and develops its berries around New Year’s time in the winter. It would
make sense to use the mistletoe, but this time in tincture form, as an extract,
in order to reengage the Ego in the body by forcing it to overcome the
“contrariness” of this herb. There is an African tradition mentioning mistletoe
therapy in diabetes. In experimental animal studies mistletoe treatment could
not show a direct sugar level lowering effect but it had a positive influence
on diabetes associated symptoms, such as hyperphagia (overeating) and
polydipsia (overdrinking).
Any form of change in lifestyle results in an increased “mastery” of the
Ego over the physical body aligns itself with the anthroposophical understanding
of treating diabetes. Ultimately, the age-old observation that aerobic exertion
lowers blood sugar is nothing more, nor less, than the reality of the Will
taking hold of the metabolic forces. Leading a rhythmical life, with structured
meals at the same time every day, is not only in harmony with the well-known
biorhythms of insulin secretion (a high and low every 15 minutes and every 90
minutes) but demonstrates the benefits when the
“I” dictates when and how
much is to be ingested.
Ultimately, the ideal will be achieved where the Ego will have the
strength to overcome its challenges through meditative-spiritual practices.
‡ Folgendes hat anthroposofische Einschlüße ‡
[Gerald Karnow, M.D.]
Diabetes is increasing. In the last decade diabetes rose by 3,3%
nationwide to 6,5% of the population. This immense increase of a chronic
illness, with its personal, social and economic consequences, poses many
questions.
We know that the biochemical problem underlying the clinical features is
an absolute or functional deficiency of insulin. This insulin deficiency leads
to impairment in the handling of body glucose (sugar), and problems in fat and
protein metabolism. Modern medical research has made, and continues to make,
ever more refined discoveries about the cause and manifestation of this
disorder which involves essentially every aspect of the human body. The fact
that we can treat diabetes with insulin, saving the lives of millions, is
gratifying to the patient and physician. But we might also ask: what is going
on beyond the biochemical problem? Could the rising incidence be a consequences
of actions on the part of society or the individual?
To me this is an important question and unless we ask it the person will
be excluded. Injecting insulin or taking medications may not be sufficient for
some.
Anthroposophical medicine does provide insights which might be helpful
in engaging the whole person as an important participant in the potential
healing or therapy of this complex disorder. In the book by Rudolf Steiner and
Ita Wegman "Fundamentals of Therapy," in the chapter on diabetes
mellitus we read that: "Where there is sugar, there is ego organization;
where sugar is generated, the ego organization appears and orients the subhuman
(vegetative, animal) corporeality towards the human." The most dramatic
demonstration verifying such a statement is the hypoglycemic (low sugar) state,
in which very low blood sugar leads to unconsciousness and ultimately, if not
replaced, to death.
In diabetes mellitus the ego orga¬nization becomes so weakened that it
can no longer effectively act on the substance of sugar. What should have
happened to sugar through the ego organization then happens to it through the
astral and etheric domains. The structure, substance, and function of all
components of our body are not at our own service but act out their own
"extra human" impulses. Our blood sugar level is not maintained
within a stable range, it acts like sugar in a sugar jar, the more you pour in
the more you have, only in the body it's not really yours - you can't use it!
Blood vessels lose their normal structure, the finer tissue components of blood
vessels overgrow, and become too `alive' to permit the ego to live properly in
the body. The body can become estranged to the extent that limbs need to be
amputated because of inadequate blood supply.
We can cite innumerable other examples showing how the body
progressively loses its function as intended for the ego because the
organization that needs to be active in the body is too weak. Hence the
question: what can be done to strengthen the ego organization?
If health experts blame the wired couch potato culture of our times and
realize that obesity is closely tied to diabetes, certainly we can realize that
we, as ego beings, are not very involved in penetrating our body as our
instrument, permitting vegetative and animal functions to predominate.
In fact, diabetes is aggravated by everything that diverts the ego
organization from an engaged function in body activity, i.e. most of our
passive-receptive activities today. It has been realized in the past, and still
is today, that good hard physical activity, work in short, has a very helpful
effect on those with diabetes or pre-diabetes.
As the body tissue used for movement becomes ever less penetrated by the
ego, as the blood, vehicle for the ego, becomes less able to individualize the
sugar, so also the nervous system substance becomes estranged from the weakened
ego organization resulting in diabetic neuropathy.
In considering these problems it is helpful again to quote Fundamentals
of Therapy: "Processes taking place in the head organization should be
parallel processes to soul and spirit activity. However, because the latter
activities take their course too fast or too slowly, they fall out of the
parallelism. It is as if the nervous system were thinking independently
alongside the thinking human being; but this is an activity which should only
be carried out during sleep. In the diabetic, a form of sleep in the depths of
the organism runs parallel to the waking state".
Of course much more should be said for a deeper insight into diabetes,
but perhaps what has been shared here, out of a spiritual scientific view
(never contradicting but always complementing and enhancing the natural scientific
view), shows that this complicated chronic illness is a mandate to the human
being—to be wake and be active in thinking, feeling, and willing, and to engage
in enthusiastic home and work activities.
In this way we can meet the illness-prone effects of our present
civilization as active agents.
Phytologie: Rezepte aus dem Papyrus Ebers
Tarax.