Herpes viren allgemein
Herpes zoster (Gürtelrose) und Folgebeschwerden (Post-Zoster-Neuralgie). Für diese Krankheit ist der begleitende Einsatz von Variolinum meistens unverzichtbar,
in Kombination mit Carcinosinum oder X-Ray.
‡ Eiterung/Reizblase/Zystitis; Akute Verbrühungen und Verbrennungen 2. Grades. Zur Begrenzung der Blasenbildung und zur Schmerzlinderung. Herpes labialis, rezidivierend
Dosierung: Akut alle 30 Min. 5 Tr. D10, im Intervall 1 x tgl. 5 Tr. D30.
Wirkt rasch/Dauer: Mit D30 über Monate ‡
For Herpes Simplex 1 and 2 (oral and genital) the same remedies. When the sore has dried up, stop dosing. Put Calendula cream on it. And by the way, Calendula cream or “Hypercal” or
Nelson’s “Cuts and Scrapes” (Calendula tincture 0.45%/Hypericum tincture 0.45%.) should be your go-to first aid cream or lotion. Hopefully you’ve got it; if not, get it!
Herpes simplex = Griebe
Hessels Grippetropfen (zur unterstützenden Behandlung von viralen Infekten und Herpes simplex);
Vergleich: Arginin (fördert Herpes simplex/enthalten in Haselnüsse/Walnüsse/Mandeln/ Paranüsse/andere Nüsse/Cacao). Staphylococcus aureus (besiedelt Haut in Impetigo/oft verbunden mit Herpes simplex).
Siehe: Nosoden allgemein
Antidotiert von: Caust. Lys. Kaffee,
Aeth-a.: Herpetic eruptions on the skin, moist in nature.
Alnus rubra: [Dr. P. Rajagopalarao, Boericke, Clarke] Ø - 3C/ulcerated mucus membranes of mouth and throat and indigestion from insufficient gastric secretion.
Phytologie: Honig 3x täglich aufbringen/Stellen mit Tempo abtrocknen und wegwerfen. Stellen sind noch 10 Tage nach Aufbrechen ansteckend
Herpes 2LHERP (RNA & DNA complex) = 2LHERP Nucleic Acid Complex No.16 By Labo'Life (30 Capsules) (LaboLife)
in preventing recurrences of genital herpes.
I usually combine most MicroImmunotherapy treatments with the patient's constitutional remedy. For genital herpes I always prescribe 2LHERP for preventing recurrences and advise external tea tree oil during any outbreaks. If there is also candida I often prescribe tea tree oil orally - 1 drop placed on a piece of sugar and swallowed.
Dreitagefieber = “roseola infantum”/= “sixth disease”.
Krankheit: 6e - 24e Monat, 3 Tage Unruhe/Bauchweh/Fieber continua/Fieberkrämpfe/nach Fieberabfall Hautausschlag beginnend im Nacken/ohne Beschwerden/spart Gesicht aus;
Antidotiert von: Lach. (Fieber continua). Acon. (unruhig/verlangt Kuhle/Exanthem hellrot wie bei Scharlach). Bell. (ergeben/verlangt Wärme/Ausschlag rot wie Masern).
Ferr-p. (WohlBEfinden/hellrotes Exanthem)
Eppstein bar o. Pfeiferisches Drüsenfieber
Herpes zoster = Gürtelrose
[Christiane Petras] Herpes zoster (Gürtelrose) und Folgebeschwerden, z.B. Post-Zoster-Neuralgie. Für diese Krankheit ist der begleitende Einsatz von Variolinum meistens unverzichtbar, in Kombination mit Carcinosinum oder X-Ray.
Siehe: Nosoden allgemein
Antidotiert von: Dol. Vario.
Arg-n (Schleimhäute/Haut/Knochen/Periost) herpetischen Kranken
Ars. brüllendes Brennen nachts
Canth. wütendes Brennen/große Blasen
Caust. ätzende Verbrennungsschmerz
Mez. wellenartiges Brennen/“Wie verbrüht“/< nachts
Merc. specific for relieving the burning, and preventing the appearance of new crops. Right side, ext. across the abdomen
Prun. Entzündung im Augenbereich/Hornhaut/Regenbogenhaut/übler SCHMERZ
Ran-b. stechendes, juckendes Brennen/Bläschen im Rippenbereich
Rhus-t. brennender Juckreiz/> Kratzen/Wärme/Bewegung/< nachts
Apis: Burning , stinging pain, large vesicles, ulcerates, starts in cold weather, < warmth, > cold application.
Ars.: Intense burning blisters, nausea , prostration, < cold, > warmth. < after midnight.
Mez.: With severe neuralgic pains, itches after scratching, brown scab forms, < from touch.
Ran-b.: Vesicles filled with thin acrid fluid. Neuralgic pain (intercostals). < touch/motion/after eating.
Rhus-t.: Probably no remedy more often found useful. Especially after getting wet.
Vario.: Burnett said Varolinum has wiped out the condition, pain and all.
----------- + brennen: Gali-a
The herpes family of viruses consists of more than 80 distinct types of viruses that are found in nearly every kind of animal that has been investigated
On going research is trying to find out whether herpes viruses cause some or all of many diseases, alone or in conjunction with other factors, or whether they occur in a secondary
association with other causes. Determining the cause of many such diseases is more difficult than it seems, because so many factors show up together when disease symptoms appear.
Overview of the 9 Types of Herpes Viruses Found in Humans:
Herpes simplex virus - Type 1 HSV-1; fever blisters/cold sores of the face, mouth, and lips
Human Herpes Virus-1, HHV-1: fever blisters/cold sores of the face, mouth, and lips/occur via breaks in the skin barrier around the mouth or elsewhere
Herpes simplex-1 causing cold sores and possibly spreading to the genital region/now linked with the development of serious neurological diseases such as Alzheimer’s disease, Bell’s palsy and trigeminal neuralgia.
Recent research also shows that coinfection by HSV-1 and HIV (human immunodeficiency virus) can enhance the activity of both viruses in AIDS and non-genital herpes lesions. HSV-1 infects about 50% of the population.
Herpes simplex virus - Type 2 (HSV-2; also Human Herpes Virus-2, HHV-2).
This type is the usual cause of genital herpes, which is classified as a sexually transmitted disease. HSV-2 reached epidemic status in the 1980s and 1990s (increased incidence among teenagers). In the world of virus classification, HSV-2 and HSV-1 are nearly indistinguishable except for their different clinical symptoms. However, even these differences are inconsistent, since both types of herpes simplex can cause oral and genital herpes outbreaks.
Herpes zoster virus (HZV; also Varicella zoster virus, VZV, and Human Herpes Virus-3, HHV-3). Chickenpox results from a first-time infection by HZV. When this virus recurs later in a person’s life, it causes shingles = Herpes zoster. As the average age of our population increases, more and more people are suffering recurring bouts of postherpetic neuralgia (nerve pain) as a result of shingles. This herpes virus is considered to be the most infectious of the known herpes viruses. More than 90% of the population is infected. HZV has been linked to the autoimmune disease called lupus. Furthermore, HZV outbreaks, which are now epidemic among people with AIDS, are often the earliest indicator of HIV infection. Epstein-Barr virus (EBV; also, Human Herpes Virus-4, HHV-4). The major
cause of infectious mononucleosis (‘kissing disease’), EBV may also be the leading culprit in causing Chronic Fatigue Syndrome and other disorders of the immune system. EBV has also been linked with lupus, lymphomas, and other cancers. This virus is now considered to be quite damaging and mutagenic in the body. Around 75% of the population will test positive for EBV.
Cytomegalovirus (CMV; also, Human Herpes Virus-5, HHV-5). CMV can cause mononucleosis and hepatitis and it can also be sexually transmitted.
Recent research suggests that CMV has a role, in conjunction with other types of viruses, in turning on cancer genes. The occurrence of CMV is strongly correlated with vascular diseases such as coronary artery disease and atherosclerosis.Even though it is generally asymptomatic, CMV may turn out to be a key factor in the development and progression of heart and blood vessel disease, the leading killer in all developed nations. CMV infects about 60% of adults, but is even more common among homosexual men and is associated with AIDS.
Human herpes viruses - Types 6, 7, 8 and 9 (HHV6, HHV7, HHV8, and HHV9, respectively). All HHVs are associated with disorders of the immune system, especially AIDS. HHV8 is also called Kaposi’s sarcoma-associated human herpes virus (KSHV), which causes a type of skin cancer that occurs most often in people with AIDS. The recent discovery of new HHVs in people with AIDS suggests that there are more to be found. Indeed, new types of these herpes viruses are probably evolving every year. HHV-6 and HHV-7, both found
in about 90% of the population, are two closely related viruses that are relatively new discoveries and are considered to be “universal” herpes viruses. Infection
with HHV-6 during childhood causes “roseola infantum” = Drei-Tagen-fieber a.k.a. “sixth disease”.
HHV-6 has recently been linked with the development of MS. Herpes viruses can occur without symptoms in nerve ganglia, which are at the bases of nerve cells near the spine or throughout the face. In these locations the viruses are seemingly in. They remain at this stage until something triggers them to move up the nerve fibers, enter healthy skin cells, and cause sores. Typically, a familiar pain or itching precedes the formation of sores, signifying an oncoming outbreak that will result in the well-known fever blisters. Sores do not always appear, though, especially in HZV infections. Internal blooms of HZV can instead cause unpleasant or even stabbing pains in nerves around the waist or spine.
HSV and HZV are trickier than most viruses because they are latent for long periods of time and because they can travel from one cell to another without ever leaving the internal environment of the cell. Both of these properties enable herpes viruses to escape notice by our immune system, at least until they burst out of the cells they have just killed and attract the attention of our antibodies. This is why our immune system is helpful in fighting herpes outbreaks but is incapable of entirely eliminating herpes viruses from our bodies.
As you can see, herpes aren’t just nuisance diseases like once thought. New research links herpes viruses not only to the well-known diseases like cold “Herpes viruses are universal in all animals/humans. The six most prevalent human herpes viruses infect 50% or greater of the global population. Actually, these numbers are low estimates since they are based on findings in serum. which can produec false negative results. Data source: Leicester University (U.K.) Microbiology Department”
sores, genital herpes and shingles, but to even more serious human diseases such as: heart disease, hypertension, Alzheimer’s disease, cancer, lupus, and multiple sclerosis. This new link is supported by the fact that all herpes viruses set-up life-long (latent) infections in people. The old logic held that, since people were not visibly sick most of the time with herpes virus diseases, these viruses must be “sleeping” during most of our lives and therefore were not dangerous to our health. New research tools and techniques that can examine our bodies in much greater detail paint a much different picture. It appears that herpes viruses are not actually “sleeping” at all. They are very active in the parts of the body that they inhabit, inflicting constant, cumulative damage to critical organs in our bodies as they replicate at a low level throughout our lives. This damage begins early in our lives, at a low enough level to not produce any noticeable symptoms in generally healthy individuals.
Herpes viruses are very sensitive to their environment. In a young person with a healthy immune system, they will attack at a very low level to avoid large-scale activation of the immune system, which could deliver a damaging blow to the herpes viruses. When the herpes viruses sense that stress, aging, cancer or physical injury has weakened the body, they accelerate their attack, just as they do if they sense that the immune system is preoccupied fighting off another infection, such as a cold or flu. This is why symptomatic herpes outbreaks often follow another illness like a severe flu, a physical injury such as surgery, or periods of emotional stress. This new way of viewing herpes viruses is important that will affect everyone’s health since herpes viruses are universal factors in human health. The medical data are very clear. Virtually 100% of the human population carries at least one herpes virus. The data also show that the majority of the human population is harboring at least five herpes virus infections.
The vast literature on herpes research underscores the challenge of understanding how this family of viruses affects our immune system. Several dozen complications are already known to be associated with herpes infections of different types, and more are being discovered each year. Many such complications, such as post-herpetic neuralgia, do not respond to antiviral treatments and can drag on for months or years after the initial infection has subsided.
“Everybody probably has herpes viruses, usually of multiple types.” They are very contagious and opportunistic, but about 60% of us have strong immunity to them. So the puzzle is, given that everyone can harbor herpes viruses all of the time, why do only some people have outbreaks? Or, more to the point, what causes herpes outbreaks? Unfortunately, nobody has satisfactory answers to these questions. But, although the factors that trigger herpes outbreaks are largely unknown, we do have a few good clues.
One clue comes from the observation that physical trauma to nerve cells, such as from a surgeon’s scalpel, will activate herpes viruses. Another clue is that herpes outbreaks are associated with several kinds of stress (cold temperatures/injury/synthetic drugs/cancer/allergies/depression/infections/unhappiness/anxiety/too much sunlight)
Herpes zoster with people over 60/Patients whose immune systems are suppressed by cancer chemotherapy, radiation therapy, and drugs used after organ transplants, by AIDS or other immunosuppressive disorders, are especially susceptible to herpes outbreaks. Indeed, herpes infections in immunosuppressed patients are the most severe, leading to pneumonia, liver disease, CMV retinitis, and other serious complications that can be fatal. Smoking, poor nutrition, and recreational drug use cause stress that is detrimental to our immune system. Hard exercise, injury, surgery, and UV radiation in sunlight and in tanning Booths, aging all slow down our ability to make the principal infection-fighting cells of the immune system. These stresses and how they affect our susceptibility to infection constitute a topic worthy of a separate book.